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However, the oncogenic transformation of normal cells is frequently accompanied by a profound remodeling of metabolic pathways [5]

First, the glutamate cysteine ligase (GCL) catalyzes the formation of a dipeptide constituted by glutamate and cysteine, and then the GSH synthetase catalyzes the addition of glycine to form GSH[38], the former being the rate-limiting step

NIAID also supports research on basic immunology, transplantation and immune-related disorders, including autoimmune diseases, asthma and allergies

The reasons for variable expression of CHAC1 in different cancers are still unclear, but it appears to be critically dependent on TP53 status